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Titel:

Helicobacter pylori infection stimulates plasminogen activator inhibitor 1 production by gastric epithelial cells.

Dokumenttyp:
Journal Article; Research Support, N.I.H., Extramural; Article
Autor(en):
Keates, AC; Tummala, S; Peek, RM; Csizmadia, E; Künzli, B; Becker, K; Correa, P; Romero-Gallo, J; Piazuelo, MB; Sheth, S; Kelly, CP; Robson, SC; Keates, S
Abstract:
Chronic infection with the gastric pathogen Helicobacter pylori significantly increases the risk of developing atrophic gastritis, peptic ulcer disease, and gastric adenocarcinoma. H. pylori strains that possess the cag pathogenicity island, which translocates CagA into the host cells, augment these risks. The aim of this study was to determine the molecular mechanisms through which H. pylori upregulates the expression of plasminogen activator inhibitor 1 (PAI-1), a member of the urokinase activ...     »
Zeitschriftentitel:
Infect Immun
Jahr:
2008
Band / Volume:
76
Heft / Issue:
9
Seitenangaben Beitrag:
3992-9
Sprache:
eng
Volltext / DOI:
doi:10.1128/IAI.00584-08
PubMed:
http://view.ncbi.nlm.nih.gov/pubmed/18519558
Print-ISSN:
0019-9567
TUM Einrichtung:
Chirurgische Klinik und Poliklinik
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