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Document type:
Journal Article; Research Support, Non-U.S. Gov't
Author(s):
Petry, A; Djordjevic, T; Weitnauer, M; Kietzmann, T; Hess, J; Gorlach, A
Title:
NOX2 and NOX4 mediate proliferative response in endothelial cells.
Abstract:
Increased levels of reactive oxygen species (ROS) contribute to many cardiovascular diseases. In neutrophils, ROS are generated by a NADPH oxidase containing p22phox and NOX2. NADPH oxidases are also major sources of vascular ROS. Whereas an active NOX2-containing enzyme has been described in endothelial cells, the contribution of recently identified NOX homologues to endothelial ROS production and proliferation has been controversial. The authors, therefore, compared the role of NOX2 with NOX4...     »
Journal title abbreviation:
Antioxid Redox Signal
Year:
2006
Journal volume:
8
Journal issue:
9-10
Pages contribution:
1473-84
Language:
eng
Fulltext / DOI:
doi:10.1089/ars.2006.8.1473
Pubmed ID:
http://view.ncbi.nlm.nih.gov/pubmed/16987004
Print-ISSN:
1523-0864
TUM Institution:
Klinik für Kinderkardiologie und angeborene Herzfehler (Prof. Hess)
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