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Title:

NOX1 supports the metabolic remodeling of HepG2 cells.

Document type:
Journal Article; Research Support, Non-U.S. Gov't
Author(s):
Bertram, Katharina; Valcu, Cristina-Maria; Weitnauer, Michael; Linne, Uwe; Görlach, Agnes
Abstract:
NADPH oxidases are important sources of reactive oxygen species (ROS) which act as signaling molecules in the regulation of protein expression, cell proliferation, differentiation, migration and cell death. The NOX1 subunit is over-expressed in several cancers and NOX1 derived ROS have been repeatedly linked with tumorigenesis and tumor progression although underlying pathways are ill defined. We engineered NOX1-depleted HepG2 hepatoblastoma cells and employed differential display 2DE experiment...     »
Journal title abbreviation:
PLoS ONE
Year:
2015
Journal volume:
10
Journal issue:
3
Pages contribution:
e0122002
Language:
eng
Fulltext / DOI:
doi:10.1371/journal.pone.0122002
Pubmed ID:
http://view.ncbi.nlm.nih.gov/pubmed/25806803
Print-ISSN:
1932-6203
TUM Institution:
Klinik für Kinderkardiologie und angeborene Herzfehler (Prof. Hess)
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