The detection of ischemia in the setting of cardiac intervention is a challenging problem. To outline the marker release caused by the intervention itself, we investigated values of patients after transcatheter aortic valve implantation (TAVI) without periprocedural ischemia. Serum values of CK, CK-MB, and TnT at baseline, 2h, 8h and on day 1-5 postoperatively were compared with those after surgical aortic valve replacement (SAVR). We differentiated a transarterial and a transapical subgroup. With the avoidance of aortic cross-clamping and cardioplegia, a lower and delayed rise of markers was demonstrated for the TAVI group compared to SAVR. In contrast to the transarterial subgroup, early release patterns and significantly higher markers were observed after more invasive antegrade implantation, the CK-MB values even exceeding those after surgery and thus suggesting that the access site should be considered in the diagnosis of periprocedural myocardial infarction after TAVI.
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The detection of ischemia in the setting of cardiac intervention is a challenging problem. To outline the marker release caused by the intervention itself, we investigated values of patients after transcatheter aortic valve implantation (TAVI) without periprocedural ischemia. Serum values of CK, CK-MB, and TnT at baseline, 2h, 8h and on day 1-5 postoperatively were compared with those after surgical aortic valve replacement (SAVR). We differentiated a transarterial and a transapical subgroup. Wi...
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